
Lyme disease is often described as an infection caused by Borrelia burgdorferi. But that explanation misses the bigger picture. What makes Lyme chronic isn’t only the pathogen—it’s the metabolic collapse of the host, especially around iron regulation and redox balance.
When Borrelia enters the body, the immune system activates nutritional immunity—a defense that locks away iron to starve pathogens.
Ferritin rises. Hepcidin surges. Serum iron plummets.
This is meant to be temporary.
But in chronic Lyme, inflammatory cytokines like IL-6 and TNF-α keep hepcidin elevated long after the initial infection.
Iron becomes trapped in tissues—starving red blood cells and mitochondria while feeding oxidative stress.
This creates the paradox of anemia on paper, overload in the body: fatigue, poor circulation, and rusting tissues that can’t heal.
Tissue iron excess quietly:
Even though Borrelia prefers manganese over iron, the iron-toxic terrain it creates weakens host defenses and sustains chronic symptoms like fatigue, joint pain, brain fog, and neuropathy.
Most Lyme protocols focus on eradicating the infection.
But the real solution begins with restoring metabolic integrity—reviving glutathione, balancing iron, and repairing mitochondrial function.
Glucoferrin® is a patented, pharmaceutical-grade complex that targets the root metabolic block driving chronic Lyme. It:
Rather than chasing microbes indefinitely, Glucoferrin® corrects the iron-driven bioenergetic bottleneck that allows chronic infection to persist.
Lyme disease is not just an infection—it’s a metabolic war.
And in that war, iron is one of the biggest players.
Glucoferrin® helps you win by restoring the balance life depends on.